primary rabbit anti- anoctamin- 1 (Alomone Labs)
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Alomone Labs
primary rabbit anti- anoctamin- 1
Primary Rabbit Anti Anoctamin 1, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/acl/Anti-TMEM16A+(ANO1)+(extracellular)+Antibody/pm42277581-77-29-38
Average 93 stars, based on 12 article reviews
Primary Rabbit Anti Anoctamin 1, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 12 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/acl/Anti-TMEM16A+(ANO1)+(extracellular)+Antibody/pm42277581-77-29-38
Average 93 stars, based on 12 article reviews
primary rabbit anti- anoctamin- 1 - by Bioz Stars,
2026-10
93/100 stars
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Immunofluorescence:Article Title: Orai1 calcium channel inhibition prevents progression of chronic pancreatitis Article Snippet: Images were captured by Zeiss Axio Imager.M2 microscope (Carl Zeiss Microscopy) with 2.5 × objective (Zeiss, NA: 0.07), 5 × objective (Zeiss, NA: 0.15), 20 × objective (Zeiss, NA: 0.8), and 40 × objective (Zeiss, NA: 0.75); they were then subsequently analyzed. .. For immunofluorescence staining, whole samples were frozen and cut at –20°C; they were then fixed in 4% paraformaldehyde (PFA) or PSCs seeded onto cover glasses were fixed in 4% PFA and labeled with primary antibodies overnight (anti–α-SMA, Novus Biologicals, NB300-978; anti-CFTR, Alomone labs, Staining:Article Title: Orai1 calcium channel inhibition prevents progression of chronic pancreatitis Article Snippet: Images were captured by Zeiss Axio Imager.M2 microscope (Carl Zeiss Microscopy) with 2.5 × objective (Zeiss, NA: 0.07), 5 × objective (Zeiss, NA: 0.15), 20 × objective (Zeiss, NA: 0.8), and 40 × objective (Zeiss, NA: 0.75); they were then subsequently analyzed. .. For immunofluorescence staining, whole samples were frozen and cut at –20°C; they were then fixed in 4% paraformaldehyde (PFA) or PSCs seeded onto cover glasses were fixed in 4% PFA and labeled with primary antibodies overnight (anti–α-SMA, Novus Biologicals, NB300-978; anti-CFTR, Alomone labs, Labeling:Article Title: Orai1 calcium channel inhibition prevents progression of chronic pancreatitis Article Snippet: Images were captured by Zeiss Axio Imager.M2 microscope (Carl Zeiss Microscopy) with 2.5 × objective (Zeiss, NA: 0.07), 5 × objective (Zeiss, NA: 0.15), 20 × objective (Zeiss, NA: 0.8), and 40 × objective (Zeiss, NA: 0.75); they were then subsequently analyzed. .. For immunofluorescence staining, whole samples were frozen and cut at –20°C; they were then fixed in 4% paraformaldehyde (PFA) or PSCs seeded onto cover glasses were fixed in 4% PFA and labeled with primary antibodies overnight (anti–α-SMA, Novus Biologicals, NB300-978; anti-CFTR, Alomone labs, Recombinant:Article Title: AAV gene therapy rescues hearing and balance in a model of CLIC5 deafness Article Snippet: .. Reagents and tools table Experimental models C57BL/6JClic5nmf318/J Jackson Laboratory #005329 Recombinant DNA ssAAV9/PHP.B -CB6-Clic5-3XFLAGP2A-TurboGFPWPRE Viral Vector Core at Boston Children’s Hospital N/A scAAV9/PHP.B -CB6-Clic5-3XFLAGP2A-TurboGFPWPRE Viral Vector Core at Boston Children’s Hospital N/A ssAAV9/PHP.B -CB6-TurboGFPWPRE_Control Viral Vector Core at Boston Children’s Hospital N/A scAAV9/PHP.B -CB6-TurboGFPWPRE_Control Viral Vector Core at Boston Children’s Hospital N/A Antibodies Polyclonal Myosin VIIa Proteus Biosciences cat# 25‐6790 ANTI-CLIC5 Alomone Labs cat# Article Title: RIPOR2-mediated autophagy dysfunction is critical for aminoglycoside-induced hearing loss. Article Snippet: In brief Li et al. find that aminoglycoside binds to and triggers rapid translocation of RIPOR2 in cochlear hair cells.. RIPOR2 interacts with the core autophagy component GABARAP and affects autophagy activation.. Inhibiting this pathway prevents aminoglycosideinduced hearing loss, and this suggests that autophagy pathway components are potential therapeutic targets for preventing aminoglycoside ototoxicity. Western Blot:Article Title: Structural heterogeneity of the ion and lipid channel TMEM16F Article Snippet: .. Anti-TMEM16B (1 citation), Human, rat and mouse, WB, IFC, LCI and IF (https://www.alomone.com/p/anti-anoctamin-6-extracellular/ Article Title: RIPOR2-mediated autophagy dysfunction is critical for aminoglycoside-induced hearing loss. Article Snippet: In brief Li et al. find that aminoglycoside binds to and triggers rapid translocation of RIPOR2 in cochlear hair cells.. RIPOR2 interacts with the core autophagy component GABARAP and affects autophagy activation.. Inhibiting this pathway prevents aminoglycosideinduced hearing loss, and this suggests that autophagy pathway components are potential therapeutic targets for preventing aminoglycoside ototoxicity. other:Article Title: AAV gene therapy rescues hearing and balance in a model of CLIC5 deafness Article Snippet: ANTI-CLIC5 , Generated:Article Title: RIPOR2-mediated autophagy dysfunction is critical for aminoglycoside-induced hearing loss. Article Snippet: In brief Li et al. find that aminoglycoside binds to and triggers rapid translocation of RIPOR2 in cochlear hair cells.. RIPOR2 interacts with the core autophagy component GABARAP and affects autophagy activation.. Inhibiting this pathway prevents aminoglycosideinduced hearing loss, and this suggests that autophagy pathway components are potential therapeutic targets for preventing aminoglycoside ototoxicity. Virus:Article Title: RIPOR2-mediated autophagy dysfunction is critical for aminoglycoside-induced hearing loss. Article Snippet: In brief Li et al. find that aminoglycoside binds to and triggers rapid translocation of RIPOR2 in cochlear hair cells.. RIPOR2 interacts with the core autophagy component GABARAP and affects autophagy activation.. Inhibiting this pathway prevents aminoglycosideinduced hearing loss, and this suggests that autophagy pathway components are potential therapeutic targets for preventing aminoglycoside ototoxicity. Subcloning:Article Title: RIPOR2-mediated autophagy dysfunction is critical for aminoglycoside-induced hearing loss. Article Snippet: In brief Li et al. find that aminoglycoside binds to and triggers rapid translocation of RIPOR2 in cochlear hair cells.. RIPOR2 interacts with the core autophagy component GABARAP and affects autophagy activation.. Inhibiting this pathway prevents aminoglycosideinduced hearing loss, and this suggests that autophagy pathway components are potential therapeutic targets for preventing aminoglycoside ototoxicity. |
![ACLY activity regulates MDR1 expression in colorectal cancer. (A) Transcript levels of ACLY and MDR1 (ABCB1) in colorectal cancer (red) and normal colon tissues (grey) analyzed using GEPIA (TCGA/GTEx datasets). (B) Immunoblot analysis of MDR1 in SW480 and DLD1 cells treated with the ACLY <t>inhibitor</t> <t>BMS-303141</t> (20 or 50 μM, 48 h). (C) Immunoblot analysis of ACLY and MDR1 in SW480 and DLD1 cells transduced with empty vector (pLV) or ACLY-overexpressing vector (pLV[Exp]-hACLY). (D) Immunoblot analysis of ACLY and MDR1 in control and ACLY-overexpressing cells treated with BMS-303141 (50 μM, 48 h). (E) Relative ABCB1 mRNA levels in control and ACLY-overexpressing cells, and in cells treated with BMS-303141. (F) Relative ACLY mRNA levels under the same conditions. Data are presented as mean ± SD (n = 3 unless otherwise indicated). Statistical significance was determined using unpaired two-tailed t-tests or one-way ANOVA with Tukey’s post hoc test. *P < 0.05; **P < 0.01; ***P < 0.001.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_2016/pmc13142016/pmc13142016__gr1.jpg)
